Participants: C.A. Murray (Host), Milton Teske, Steven Bohr
Series Code: CVNU
Program Code: CVNU000004S
|
00:10 Hello, and welcome to SumTV's Coronavirus Report.
00:14 This is our weekly update on the happenings with the coronavirus. 00:18 With me is Pastor Stephen Bohr. 00:20 - Pastor, good to have you here. - Good to see you. 00:22 I'm C. A. Murray. 00:23 And our resource person happens to be the chairman of our board; 00:26 that's Dr. Milton Teske. 00:28 Good doctor, we know you're busy, so we're glad and thankful 00:32 that you've been able to take out a few minutes of your 00:35 very busy schedule to be with us. 00:36 Glad to be here today. 00:38 He's got so much information, he's got a couple of new things 00:41 he wants to share with us today. 00:43 So we're going to just launch out and start him 00:46 because we've got a lot to talk about 00:47 and not a lot of time to do it. 00:48 Pastor, I know you've got a couple of questions, 00:50 so I'm going to put it in your hands, 00:52 and then I've got a couple. 00:53 Then the good doctor has some new information for us 00:56 that he wants to share. 00:57 Okay, very good. 00:59 One thing I've really admired about you, Dr. Teske, is your 01:03 vast knowledge of physiology. 01:06 Besides being a medical doctor. 01:08 And I understand that this virus works differently 01:13 than other similar viruses. 01:15 So could you share with the audience, you know, 01:18 the differences in the way that this virus works 01:21 versus SARS and some of the others? 01:23 Yeah, that's really on the front edge of what 01:26 we're discovering now, because this virus is not just 01:30 another coronavirus. 01:32 It's not just a SARS virus with slightly different infectivity. 01:38 Although, it is more infectious. 01:40 They found out that a SARS virus, when it replicates 01:44 and destroys a cell, one virus will make 01:47 10, 20, 30 more viruses. 01:49 With this COVID virus, it's more than 100. 01:53 So we've got a whole order of magnitude of just 01:56 more virus particles coming out faster 01:59 in the infection process. 02:02 But the really scary stuff we're finding out here 02:06 is a totally different area of function that this COVID virus 02:11 has than its closest relative, the SARS virus. 02:15 You know, there's the SARS and MERS, 02:17 and all these other different types of coronaviruses, 02:21 plus, a lot of common coronaviruses that just cause 02:23 light common cold symptoms. 02:26 But this one has really been engineered, designed. 02:31 It's built of what the virology scientists and researchers 02:36 call, enhancement of function. 02:39 In other words, it's able to do something different 02:43 that it couldn't do before. 02:46 And what we're discovering, one of the big things, 02:49 is its effect on the body's immune system. 02:56 You know, when you look at the virus, it's got these 02:58 little spikes out on the side, and those little spikes 03:01 is how it infects. 03:03 And those little spikes, they attach and attack 03:07 an enzyme, angiotensin-converting enzyme. 03:11 Or they just often refer to it as ACE2. 03:14 And this enzyme is on the surface of many cells. 03:19 It's particularly prominent on the epithelium in the lungs. 03:22 That's why when it gets down in the lungs it causes 03:24 these really bad pneumonias and you end up on ventilators 03:28 or ECMO or something like that trying to keep you alive, 03:31 because it's doing so much lung damage. 03:34 But there's also, these ACE enzymes are on the myocardium. 03:40 And that's why the virus can attack the heart 03:43 and cause a bad myocarditis, destroy heart function, 03:47 and people go into cardiogenic shock, 03:49 and maybe half the people die from heart failure there. 03:54 But the new thing that we're discovering is its effect on the 03:59 body's immune system. 04:01 Now when doctors follow people for infections, 04:06 one of the most common tests they do is a CBC 04:09 where you count the blood cells. 04:11 And you have the different types of white blood cells in there. 04:16 And there's the neutrophils, and the lymphocytes, 04:20 and the eosinophils. 04:21 Well, the lymphocytes is one big component of that. 04:25 There's you T lymphocytes and your B lymphocytes. 04:28 But even those are subdivided. 04:30 It's a very complex military that your body has designed 04:34 to fight off the enemy. 04:36 And there's very specialized functions between these 04:38 different cells and what they do. 04:41 But interestingly, in these COVID patients 04:45 not only do we not get a big high elevated white count 04:48 like we do in a bacterial infection, 04:50 the white count doesn't seem to elevate, 04:52 but particularly the lymphocytes get lower. 04:58 We use the word, lymphopenia, which just means 05:01 there's too little lymphocytes. 05:03 And we're finding that particularly in bad cases 05:06 the lymphocyte count goes way down. 05:08 And of course, the lower your lymphocyte count gets, 05:11 the more likely you are to succumb to this disease. 05:17 If you're somewhat medically minded or just kept up on 05:22 some things, you probably are aware that we're talking 05:26 something similar to HIV in a sense. 05:28 Because what does the HIV virus do? 05:31 The AIDS virus actually attacks and destroys 05:35 your body's immune cells. 05:38 And when it destroys enough of them, 05:40 there's no way for you to fight off the infection. 05:44 So one of the really important cells here, the T lymphocytes, 05:49 some of them are, we use the term, NK's, natural killers. 05:54 Or sometimes we refer to them as killer T's. 05:57 These particular cells, they're not the ones 06:00 that make antibodies. 06:01 That's the B lymphocytes. 06:03 But these T lymphocytes, these killer lymphocytes, 06:06 Their job is to actually kill and destroy cells. 06:13 Like, for instance, the cancer cell. 06:15 And a lot of our new cancer things, we're trying to somehow 06:19 get them to be better at targeting cancer cells. 06:23 Many people are beginning to realize that cancer is really 06:28 a failure of your body's immune system to recognize and destroy 06:32 cancerous cells that are popping up all of the time. 06:35 Your body recognizes them, destroys them, and you go on 06:38 and never knew there was anything there. 06:39 I mean, cancer has got to be billions of cells big before 06:42 you can, big enough for you to find and realize, 06:45 "Oh dear, I've got cancer." 06:46 By then it's so far down the road, things are often 06:48 much more difficult to fight. 06:50 But back to our story here. 06:52 These killer T's, their job is to identify an enemy. 06:56 But not just cancer cells. 06:58 When a virus infects a cell, we talked about this in our 07:02 first episode here, but when the virus infects the cell, 07:06 it attaches to it and it injects its viral code inside the cell. 07:11 It just hijacks it, takes it over, and now it is all focused, 07:16 instead of building the various components that the cell needed 07:20 for whatever type of cell it was, now it's building 07:23 viral components. 07:25 And assembling inside there, actually it's assembling virus 07:29 particles in there, and we're getting viruses. 07:31 And like we just mentioned, in this case it will build over 07:34 a hundred more COVID viruses before the cell is 07:39 finally so overwhelmed that it breaks down and releases 07:42 those viruses. 07:45 Well killer T's can go recognize that this cell is infected. 07:50 They can tell by the changes in the surface 07:52 the cell is infected. 07:53 And what a killer T is, it goes to the surface of one of these 07:56 infected cells and it actually makes a hole in it. 08:00 It's like it has the ability to just punch a hole. 08:05 Now how do you make a hole in a cell membrane? 08:06 It's actually got special little protein rods, 08:09 and it just spshh, like a machine gun. 08:11 It shoots a barrage of these little rods into the side. 08:14 These little rods, they lock together, each together 08:17 in a ring making a tube of these little rods. 08:21 And this tube is now a hole. 08:24 And so, now you've got a hole. 08:26 And it's like you took a machine gun and shot the 08:28 side of this cell full of holes. 08:30 Well now we've got no integrity between 08:32 the inside and the outside. 08:34 But even more than that, the killer T actually injects 08:37 substances in there like poisons, toxins, 08:40 that actually destroy the cell. 08:42 They destroy the viruses in there, they destroy the cell. 08:45 They wipe it out. 08:46 I mean, once a cell is infected, it's doomed. 08:49 There's no way you're going to save that cell's life. 08:51 But for the sake of the body, we need to get rid of that cell. 08:55 And killer T's are really good at doing that. 08:58 And so, you might say killer T's are the number one enemy 09:03 of the COVID virus and the warfare that's going on there. 09:08 Well now, here is what we've discovered 09:12 about the COVID and the killer T's. 09:17 And that is, a COVID virus has the ability 09:20 to go to the surface of a T lymphocyte 09:24 and actually fuse with the surface. 09:28 And when it fuses with the surface, 09:30 it actually injects itself in there 09:32 and it actually can take over the cell and kill it. 09:37 And interestingly, it's different than the way 09:42 it attacks the lung epithelium and the myocardium, 09:45 and all these other parts. 09:47 Because in most of these cases, like we talked about, 09:50 it attacks, those little spikes, they attach at that ACE enzyme, 09:54 angiotensin-converting enzyme. 09:57 And as they attach there and inject their stuff 10:01 and take over the cell, that's how it works. 10:02 But in this case, T lymphocytes have very few of these 10:08 particular enzymes on their surface. 10:09 They're not really involved in that function. 10:12 And what happens is, these spikes have now been modified 10:16 in such a way that they come up to a T lymphocyte surface, 10:21 they actually fuse with the surface, 10:24 just merge right into the surface, 10:26 and release everything into the surface 10:28 breaking down the actin fibers, the things that give integrity 10:34 underneath your cell membrane. 10:36 Just open it wide open and it releases its material 10:40 inside the cell, and it actually leads to the death 10:44 of the T lymphocyte. 10:46 Although, interestingly it doesn't take over and 10:49 make more viruses in the T lymphocyte. 10:52 In other words, it's not using the T lymphocyte 10:54 as a factory to make more COVIDs, 10:58 but it destroys the T lymphocyte in this process. 11:02 I mean, in other words, somehow we've added the function to this 11:08 to come and fuse with that. 11:09 And this is a new function. 11:11 No other coronavirus can do this. 11:14 The worst one we had before COVID was the SARS. 11:18 And the SARS virus cannot fuse with the T lymphocyte. 11:21 And the MERS virus cannot fuse with it. 11:25 And the other coronaviruses. 11:26 This is what the researchers, the virologists call, 11:30 enhancement of function. 11:32 In other words, we have now altered the surface of this 11:35 with various sequences in the gene which changes the protein. 11:40 And now we've added to the function to fuse and destroy 11:44 T lymphocytes. 11:46 Now HIV viruses can fuse and destroy T lymphocytes. 11:52 Ebola viruses can do that. 11:55 But coronaviruses have never done that 11:58 before this particular version. 12:00 So we have now added a whole new dimension to pathology 12:06 and disease here. 12:07 You know, we've taken something really evil and bad 12:12 and we've spread that evil now into this very infective virus. 12:18 So it's very infective, but it's also becoming 12:21 more lethal by adding this particular function to it. 12:25 It's part of the, you might say, the scariness of what's 12:28 going on with this particular disease. 12:31 And it matches some of the stuff we see. 12:33 Not just in the fact that we see lymphocytes going down, 12:37 but we see people responding, you know, with destruction 12:40 of their body's immune system, you know. 12:42 When they do an autopsy, they say it not only just 12:45 destroys, you know, the lungs or the heart, these others, 12:48 but we're finding out the body's immune system 12:50 is really damaged in severe cases of this right here. 12:54 And we've just made people more susceptible. 12:58 The strength of this, you know, for those who thought and said 13:03 that this was pretty much just like a flu or common cold, 13:07 this is a flu with a machine gun in its hands. 13:11 Because it has this other wrinkle to do some things 13:15 that these others don't do, attacking the T cells and 13:18 tearing down the body's immune system. 13:20 That's a whole other ball game. 13:21 Right, that's a whole different level of evil 13:26 that has come in here, the pathology. 13:28 It's just really, its ability to do that, how that got designed 13:35 and built into there is really an awesome and scary thing. 13:39 But it explains some of the stuff we see. 13:43 You know, you'll see somebody that very rapidly 13:46 will go from being a very healthy individual 13:50 to become very sick and in the hospital in ICU, 13:55 and they die from it. 13:57 And while the majority of those we see that go in there 14:04 are older people with more compromised, you know, 14:07 other things already wrong with them. 14:09 They may have a bad heart or lungs, or something there. 14:12 Not all of them. 14:14 There are also young healthy people with apparently 14:17 nothing wrong with them that would make them 14:21 more susceptible to this. 14:23 That's what I see that is a little frightening; 14:25 this idea that young healthy people are going from 14:28 relatively good health to at death's door 14:32 very rapidly once they've been diagnosed. 14:36 So it seems as though this virus has the ability 14:40 to seek out whatever ails you and magnifies that 14:44 and complicates that, and makes this bee line 14:48 to, you know, morbidity. 14:51 Because whatever you have, it will find it 14:53 and it will work on that. 14:55 Yeah, any part of our system that's broken down is going to 14:59 make us more susceptible to this particular virus. 15:03 But in these cases where we get what appears to be a 15:05 young healthy individual, the vulnerability 15:09 probably is genetic. 15:11 There is probably some genetic variation 15:14 and some gene coding for some protein 15:18 on some cell that is now very susceptible 15:23 to this virus, and suddenly this person is actually 15:26 very susceptible to this particular virus. 15:30 Otherwise, in life they would have no problem 15:32 because that particular, you know, genetic change 15:36 probably is no big deal because everything else works fine. 15:41 But now in the presence of this virus, suddenly 15:44 it takes advantage of that one small weakness 15:47 and we now have a person that was totally healthy 15:52 and now very rapidly the disease progresses 15:56 and they die for it. 15:57 We don't know what particular one it is, 16:00 and maybe there's several different genetic errors 16:05 or changes in there that can make one more susceptible. 16:08 You know, this virus is so new the scientists are rushing 16:11 to try to discover this cell stuff. 16:14 Every day we're getting new... 16:16 The stuff I'm just sharing with you just came out this week. 16:19 We couldn't have had this lecture last week because 16:22 that wasn't even known then. 16:24 And so who knows what we'll find by next week that comes out. 16:29 But scientists are rapidly working on this 16:31 all over the world and new little facts and details 16:35 and nuances are coming out. 16:36 And, you know, it's really a very developing thing 16:41 right here that we're on the front lines of 16:43 to figure out what this is, what it can do, 16:45 and of course most importantly, what can we do about it? 16:50 I have a couple of questions. 16:52 You hear a lot about plasma therapy. 16:55 So is that kind of like calling in the reinforcements? 16:57 Using the plasma of somebody that was infected and overcame 17:00 the disease, and now they inject those into another person's 17:04 immune system to kind of bring in the reinforcements? 17:07 Yeah, yeah, when you take somebody's plasma, 17:10 you go to the blood bank, they draw off your blood. 17:13 They separate out the red blood cells and you get the 17:16 serum, the clear yellow part of the blood, 17:18 the plasma that's left here. 17:20 Well, in there is antibodies. 17:23 Antibodies are special proteins made by 17:26 your body's immune system. 17:28 It's a complex thing of how they know what to make, 17:30 and how to make it, and who to make it, 17:32 and to make it. 17:33 But the B lymphocytes are the ones that automatically 17:36 ultimately turn into the factories 17:39 to manufacture antibodies. 17:43 Antibodies are actually shaped like the letter "Y". 17:46 There's a central part, and then these two arms. 17:49 And both of the two arms are identical, and they've got sites 17:52 that specifically can identify a particular virus or bacteria. 17:57 In this case, the COVID virus. 17:59 And they can lock onto it. 18:01 But because there's two arms, it can lock onto two of them. 18:04 And another one can lock onto two. 18:06 And you start getting all of these viruses 18:09 stuck together in a big clump. 18:12 Well, now that they're stuck together in a big clump, 18:15 they can't actually just go out and start infecting other cells. 18:19 Now they're all kind of, sort of locked up in this. 18:21 It's like, take a bunch of criminals, 18:23 put handcuffs on them, and you handcuff them all together. 18:26 And now they're all handcuffed together, 18:29 and you've got 30, well how much crime can you do 18:32 when you're handcuffed to 30 other criminals 18:33 and you can't move your arms. 18:36 But that's just the beginning of it. 18:39 This big, the other, the tail of the "Y" 18:42 that doesn't lock on, well it's got all types of 18:45 other things that are locked onto 18:49 or that are sensed by other cells. 18:50 Now one of the important cells in your body's immune system 18:54 is the macrophage, the big eaters. 18:56 These are the ones that come out with a 18:58 process we call, phagocytosis. 19:00 They just swallow up and go at this. 19:02 Well these little antibody tails sticking out there, 19:06 it's sort of like sugar coating. 19:08 And when a macrophage finds some of these sugar coated 19:13 viruses all stuck together, you know, caramel peanut clusters, 19:18 let's go for this. 19:19 And it just moves around it, pulls it in. 19:23 And once it gets inside, then he's got... 19:26 It's inside of a little cage in there. 19:28 It's not just loose inside, it is in this little cage in there. 19:32 He brings another little cage over, a container, that's full 19:36 of powerful proteolytic enzymes and peroxidases, 19:39 and it totally oxidizes and rips the viruses and everything 19:43 in there to shreds, totally destroying it. 19:47 And so, these antibodies are important. 19:50 And if you have had the infection and your immune system 19:53 has worked properly, and your immune system has 19:56 made lots of antibodies and won this battle, 20:00 now we can take your blood that's full of these antibodies. 20:03 And now I can infuse that into you that just got infected 20:07 and really wanted to win this battle, but was having a trouble 20:11 right now, and those antibodies start locking up 20:15 all of these guys, and we can really get ahead. 20:17 So it's a really important tool where we are 20:21 working with the body's immune system, 20:23 and it's a way that you can help him fight and win this battle. 20:28 You've been victorious over it here with your antibodies, 20:31 and now you can share your antibodies with him 20:33 and help him win that battle. 20:36 So yeah, that's what plasma therapy is. 20:38 I understand it a lot better now. 20:41 I'm glad. 20:42 The other thing I want to ask is, there's a lot of discussion 20:47 these days about hydro... 20:51 ~ Hydroxychloroquine. 20:53 Yeah, that's a mouthful. Hydroxychloroquine. 20:55 ~ Yeah, hydroxychloroquine. 20:57 And it seems to be more of a political thing 21:00 than anything else. 21:01 So what is the truth about hydroxychloroquine? 21:04 Okay, hydroxychloroquine is a drug that's been around 21:07 for a long time. 21:08 We've used it for treating malaria. 21:12 For decades we've been using it. 21:14 It works much better than quinine, 21:16 which use to be the only thing we had to treat malaria with. 21:19 That really caused a lot of side effects. 21:20 Yeah, well this is much better at fighting it. 21:23 Chloroquine, and now the hydroxychloroquine. 21:26 I spent some time in Zambia, and I took it 21:30 so that I wouldn't get malaria. 21:32 Because I was in a bad area and there were mosquitoes 21:34 where there was malaria. 21:35 And I didn't want to get malaria, so I took this 21:37 and I never got malaria. 21:39 But if I had gotten bit by a mosquito, 21:41 which I got hundreds of mosquito bites 21:44 while I was there, if some of those had malaria in them, 21:47 well the hydroxychloroquine killed the malaria, 21:49 and so I never had a problem with it. 21:52 They found that it seems to work in fighting 21:55 this particular virus. 21:57 And I can't tell you the mechanism; I'm not sure 21:59 if anybody knows the mechanism by which it works. 22:02 But they have tried it. 22:03 It has worked sometimes in the past on other viruses, 22:06 so they thought, "Well, let's try it." 22:07 And it seems to work pretty good. 22:09 They were doing that in China for a while. 22:11 The nice thing about this is, it is not a new experimental drug. 22:14 This is a drug that has been around for decades, 22:17 we know its side effect profile, and it's extremely minimal. 22:20 You know, most people take it and there's no side effects 22:22 or problems at all. 22:24 And it seems to work really well. 22:27 And it's never been, quote, "officially approved" by the FDA 22:33 for fighting COVID because we never had COVID to fight 22:37 before the last couple of months. 22:38 So, has there been long-term studies, double blinded studies 22:43 with placebos and everything to prove that it works? 22:46 Well, no, there hasn't been. 22:49 And so, politically there's, you know, a battle between 22:53 those who are saying, "No, no, it's experimental. 22:55 We need to hold it. 22:56 Well, last ditch effort you can give it to people in the ICU 22:59 that are really sick, but you can't give it to everybody." 23:02 And others of us are saying, "Wait a minute. 23:04 Why don't you give this to people that are just coming down 23:07 with minor symptoms, get rid of it, get rid of the infection, 23:12 and actually save them from ending up... 23:14 Why do they have to get sick and almost die in the ICU 23:17 before we can give them this medication 23:19 that seems to work quite well?" 23:21 It certainly should be an option that's out there 23:24 for everyone, in my opinion. 23:26 Everyone can make their own decision whether they 23:28 want to take the drug or not. 23:30 But I really think that to tie it up, lock it up, 23:35 you know, is probably not appropriate. 23:38 But there's two political sides, and each have their 23:42 underlying political motivation. 23:44 And I guess that's not for us to solve today. 23:46 I want to go on record on television that if I 23:49 get seriously ill, I want it used on me. 23:52 At least pass on the word. 23:54 The problem is, you're in California. 23:58 I'm moving elsewhere. 24:00 There are other states that would be better to be in 24:02 if you would like some hydroxychloroquine 24:04 than California. 24:06 Take me elsewhere. 24:08 Is it available? Is there a... 24:10 I know it's been around for a long time, but 24:12 are there large amounts of it around? 24:14 Is it being used actively now for something else 24:16 that would pull away from that if we began to use it for this? 24:20 There are a couple of diseases that it works quite well for. 24:24 And so they're saying, well, don't everybody take it 24:26 because now these people won't have anything 24:28 for their particular condition. 24:31 But on the other hand, there are still millions of doses 24:34 stacked up on a federal level and the federal stockpile, 24:37 which they are putting out to the states. 24:40 But the states have the authority to decide 24:43 how that's used in their state. 24:45 - Or if it's used. - Or if it's used. 24:47 And how restrictive they want it to be. 24:49 And you know, I'm not going to try to solve that problem today. 24:53 But yeah, so it's the distribution of the large, 24:58 what we call, the strategic stockpile. 25:01 We have a strategic stockpile not only of this drug, 25:05 but of many different antibiotics and essential drugs 25:08 that could be used in pandemic type situations. 25:11 Stored and maintained by the federal government. 25:14 They're actually stored in Atlanta. 25:16 Some of them are stored actually in places otherwise, 25:19 in warehouses. 25:20 And they can ship out at a moment's notice 25:22 these large amounts of these medications. 25:24 And we do have those and we can ship them out, 25:27 and they are shipping them out. 25:28 But whether you'll be able to get a prescription 25:32 when you need one, I can't predict. 25:34 We'll go across the border. 25:37 And hopefully they'll have some too. 25:39 How are we doing, Dr. Teske, with personal protective 25:45 equipment, as far as that getting out, 25:47 as far as people getting sufficient numbers? 25:49 There was a lady on the other day who had been at 25:51 the Ebola breakout in Africa. 25:55 And she was saying the difference between 25:57 that and this, one of the differences is that 25:59 they had sufficient supplies of equipment. 26:02 And they were still a little short. 26:04 She was a nurse. 26:05 In fact, she was in charge of a team of nurses. 26:07 She's still running out of supplies. 26:09 How are we doing with that? 26:10 Of course, the difference between this and Ebola, 26:12 we had several small countries in Africa that needed it 26:16 for their clinics there. 26:18 Here, we've got a large nation, actually an entire world 26:22 that is suffering under this pandemic. 26:25 And every hospital in this world wants enough to protect 26:28 their healthcare workers. 26:30 And this rate of usage of protective equipment 26:34 has never been used on a worldwide scale before. 26:37 So there isn't enough. 26:39 And they're gearing up the factories, 26:41 we're putting out, you know, how many millions of masks 26:44 per day, but it's still going to be a while until they catch up 26:48 to the need. 26:50 In hospitals there are strategies for reusing 26:53 a lot of this equipment. 26:54 They're to be cleaned and sterilized and reused. 26:57 And they're doing that to try to extend its life. 26:59 In terms of what common people are going to be thinking about, 27:04 though, is you can get reasonable protection 27:07 with a paper mask or one of these cloth sewn masks. 27:10 You can wear those. 27:11 If everybody in our country started wearing these cloth ones 27:14 that you sewed made out of old t-shirts and handkerchiefs 27:18 we could stop this virus in its tracks. 27:20 Because that's enough to stop the virus spreading from you. 27:23 And if everybody put one on, well who could spread it? 27:26 It's the ones that aren't wearing that is the problem. 27:29 Good point. 27:30 ~ I wear one. ~ Alright. 27:33 So do I. 27:34 Pastor, we're just under 30 seconds. 27:38 Give us some words to give us some encouragement 27:40 for this week. 27:43 Well, this too will pass. 27:45 Lord willing. 27:47 And you know, the main thing that I found 27:50 is there's a lot of complaints about people being lonely. 27:53 But don't be lonely. 27:55 Because the Lord Jesus is near. 27:58 ~ And He's our best Friend. ~ Amen, amen. 28:00 That's a good word. 28:02 He'll never leave you, nor forsake you. 28:03 We'll see you again next week on Coronavirus Report. |
Revised 2020-05-27