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Series Code: MH
Program Code: MH240001S
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00:01 Forensic science exists to answer the
00:03 question of who done it. 00:05 Who's at fault for a particular incident? 00:07 What caused injury? 00:08 Looking for clues to find answers and solve mysteries. 00:11 Well today we're going to do a little physiologic forensics, 00:14 gather some evidence of what's behind the explosion 00:17 of chronic health problems. 00:19 Even more specifically, why is over 1/3 of the adult population 00:23 experiencing pre-diabetes or type 2 diabetes? 00:27 I'd like to know. Wouldn't you? 00:48 A detective solves problems, usually crimes. 00:52 They gather evidence to support decisions that need to be made. 00:55 Their primary object is to identify perpetrators, 00:59 to establish motives, and present a compelling case 01:02 for prosecution. 01:04 As you can imagine, they use a variety of skills and techniques 01:07 to conduct their investigations. 01:09 They meticulously analyze crime scenes, gather and analyze 01:13 forensic evidence, interview witnesses and suspects, 01:17 and collaborate with other experts 01:19 like forensic scientists or psychologists. 01:22 They need to possess excellent problem-solving abilities, 01:25 critical thinking skills, with attention to detail 01:28 to connect the dots and unravel the mysteries 01:31 surrounding criminal acts. 01:33 Detectives often work in high-pressure environments. 01:36 We may not know any personally, but they play a valuable role 01:40 in society, maintaining law and order. 01:42 Their expertise in gathering evidence, and conducting 01:46 interviews, and analyzing information 01:48 is essential in building strong cases that can stand up in court 01:53 with the goal of protecting people's rights and ensuring 01:57 that people make decisions based on the truth. 02:01 I learned that detectives specialize. 02:03 They can specialize in homicide, fraud, cyber-crimes. 02:07 There are narcotic detectives. 02:09 Some specialize in finding missing persons, 02:12 or financial crimes. 02:14 There are even pet detectives like for recovering missing pets 02:18 and tracking down animals. 02:20 I entitled this program, Physiologic Forensics, 02:23 because today we're going to be looking at 02:26 gathered evidence to help us solve for ourselves 02:29 why we may be struggling with our health. 02:33 The goal of this information is not to tell you how to live, 02:37 so much as to provide evidence that you can use 02:40 to make better decisions for yourself. 02:42 We hear quite often how type 2 diabetes is exploding. 02:46 Statistics actually reveal... 03:00 Compare that with the incidents of pre-diabetes and 03:03 type 2 diabetes in the 1960s where it was more like 1 to 2%. 03:08 That is shocking. 03:10 What's going on? 03:12 How can we change the statistics for ourselves? 03:15 Today we're going to begin investigating this topic. 03:19 It won't be exhaustive, but it will get us thinking 03:22 and help us solve the mystery. 03:25 You know, there were clues a hundred years ago as to 03:30 what we might have expected to happen in the 20th century, 03:33 and what did happen in the 20th century, 03:35 with the explosion of chronic diseases like type 2 diabetes. 03:40 Clues? Hmm. 03:42 Like, maybe the changes we've seen in how we produce 03:44 food in the modern world? 03:46 The massive infiltration of refined grains, fats, and sugar, 03:49 animals being farmed dramatically differently 03:52 than they've been for millennia? 03:54 The dumping of chemicals in our food supply? 03:57 We'd find a lot of clues if we followed the money trail; 03:59 like the subsidizing of certain products making them inexpensive 04:03 and easily obtainable through fast food. 04:05 Hmm. 04:08 I always like to help my patients to understand 04:11 what's really going on in the core, at the core 04:14 of type 2 diabetes. 04:16 In fact, what's happened in the system in our body 04:18 before type 2 diabetes that led to it. 04:21 And so, that's insulin resistance. 04:23 The driving mechanism of developing type 2 diabetes 04:28 down the road is insulin resistance. 04:31 Have you heard the term, insulin resistance, before? 04:33 In order to understand what that means, 04:36 we're going to first identify what insulin is. 04:39 Insulin's job: so what is insulin? 04:41 It's a hormone that come from the beta cells of the pancreas. 04:45 Those cells are highly specialized to recognize 04:48 what's happening with the blood glucose moment by moment. 04:51 Is it rising? Is it falling? 04:52 I think of it as, the pancreas gland knows that its 04:56 brother and sister organs need glucose to absorb. 04:59 Again, it's a gland that's ready to give 05:01 to the system that it belongs to. 05:04 So as we eat a good healthy meal with some good complex 05:07 carbohydrates, our blood sugar rises. 05:09 As it should. That's normal. 05:11 The intent there is to supply glucose to our tissues, 05:14 our muscle, and other organs. 05:17 It turns out, our cells are designed to burn glucose 05:20 as their primary fuel. 05:21 That really is the preferred fuel; 05:23 energy source for our cells. 05:26 So carbohydrates are not a bad thing. 05:27 They're a wonderful thing. 05:29 And we need carbohydrates in our diet because 05:31 we need glucose for our cells. 05:34 Alright, we have our meal, our blood sugar rises; 05:37 the pancreas gland recognizes that fact. 05:40 The beta cells recognize, "Hey, blood sugar rising. 05:42 I'm going to submit some insulin to the bloodstream 05:45 for my brother and sister organs to utilize. 05:47 They're going to need it to absorb that glucose." 05:50 Sometimes we say that glucose is the best source of fuel 05:56 for the cell; it needs to get into the cell. 05:57 But a glucose molecule is a polite molecule. 06:00 It won't go into the cell without an invitation. 06:03 Normally when we are healthy, the insulin is working properly. 06:09 We eat a meal, our pancreas will produce insulin, 06:14 and the insulin will, as a gentleman opens the door 06:18 for a lady to come in, so insulin opens the door 06:23 for glucose to come into the cells 06:25 and do all the metabolic magic. 06:27 Do all the cooking. 06:29 Dr. Kahleova just likened insulin to a gentleman who 06:32 opens doors for others. 06:34 I also like to use the analogy of the Oregonian gas attendant. 06:37 I lived in Oregon for many years, and when I lived there 06:40 it was a law that you didn't pump your own gas. 06:43 Nope, if you wanted gas in your car, the gas attendant 06:46 came to the car, and opened the gas tank, 06:48 and pumped the fuel into the tank for you. 06:51 You never had to get out of the car. 06:53 Now in my analogy, the gas is your blood glucose 06:56 and the insulin is the gas attendant that opens the tank 06:59 and puts the fuel in. 07:01 So track with me. 07:02 In order to get blood sugar out of the blood and into the cells, 07:06 insulin has to open the door, as a gentleman, 07:09 open the gas tank, as the Oregonian gas attendant. 07:13 And when insulin opens the gas tank, blood glucose 07:15 can enter in, and once inside the cell, it's combusted 07:19 and produces energy. 07:21 Energy for that cell to perform all of its activities. 07:24 And we use these analogies to help us understand 07:26 in a simple way how it all works. 07:28 But there's more. 07:31 What happens is, on the surface of our cells, we have these 07:35 receptors, and they're kind of like antenna. 07:37 They sit out on the surface of the cell and they receive 07:39 signals from elsewhere in the body. 07:41 The cell wants to know what's going on around it. 07:43 It's part of a system. 07:44 The insulin receptor is designed to receive the insulin signal. 07:49 They sit on the cell surface and they have a 07:51 portion that's outside the cell, 07:53 and then they cross through the cell membrane to the inside. 07:56 So it's like an antenna on the outside and on the inside. 08:00 I hope this isn't getting too in the weeds. 08:04 When an insulin molecule finds its way and is bound by 08:09 the insulin receptor, it's like a hand and glove fit. 08:12 There's no way this isn't designed. 08:13 They're made for each other. 08:15 Insulin binds its receptor, it actually stays on the 08:17 outside of the cell, but on the inside of the cell, 08:20 that receptor, at the moment insulin is bound on the outside, 08:24 the inside, like an antenna on the inside, 08:26 sends a message to the cell that says, 08:28 "Hey, cell, we've got some insulin out here. 08:30 You should absorb some glucose." 08:32 That's basically the message. 08:34 When everything is working well, the cell will respond 08:37 to that signal by opening up these, they're called 08:40 glucose transporter molecules. 08:42 They're openings, basically, in the cell membrane. 08:45 Again, perfectly designed for glucose molecules 08:48 to come through. 08:49 Everything is very specific in design. 08:53 Insulin brings the signal, the cell responds, 08:56 invites glucose in, glucose comes into the cell. 08:59 It yields its energy to the cell, it's broken down. 09:02 That energy winds up in the mitochondria 09:06 for it to be transformed into ATP. 09:12 In that way, the blood sugar, 09:13 the blood glucose level is regulated. 09:16 It doesn't go too high. It also doesn't go too low. 09:18 The cell gets the energy it wants. 09:21 And the muscles stay strong and the body works well. 09:24 Okay, so did you catch all that? 09:26 Just to say it again, you can think of cell receptors 09:29 like designated parking places that line the cell membrane. 09:33 Dr. Racine said that insulin in its specific parking space, 09:37 or receptor, are made for each other. 09:39 Now when insulin docks into its parking space 09:41 it sends messages into the cell through the antenna 09:45 that causes the cell to open up the gas tank 09:48 for glucose to enter and allow its energy to be 09:51 transformed into the kind of energy that the body can use. 09:55 It's wonderful how the body was designed to work. 09:58 Very wonderful. 10:00 So we've just described insulin sensitivity. 10:03 The way things are supposed to work. 10:04 And it doesn't take very much insulin to make that happen. 10:07 If you're insulin sensitive, your pancreas 10:09 puts a little bit of insulin. 10:11 Just as much as what is needed. 10:12 Not very much. 10:14 So it was just described how things are supposed to happen 10:17 when we are insulin sensitive. 10:20 Insulin resistance is the opposite. 10:22 Start picturing a gas tank lid the gas attendant can't open. 10:27 It's stuck. 10:29 Insulin resistance is a condition where our body, 10:33 specifically the brain cells, or any cells for that matter, 10:38 has the inability to consume glucose 10:43 as the primary energy source. 10:46 Insulin resistance then is a problem that's going on 10:49 inside our cells in which our cells, and I'm speaking here 10:53 first off primarily about our muscle cells, 10:55 our skeletal muscle. 10:58 Which is where most of the glucose from our bloodstream 11:00 winds up; our muscles absorb it more than any other organ. 11:04 Our skeletal muscle is the main destination for most of the 11:09 glucose in our bloodstream. 11:10 And so, it's what happens in the skeletal muscle 11:14 way back before diabetes develops 11:15 that eventuates or leads to type 2 diabetes. 11:19 It's been going on for probably years, maybe decades, 11:22 prior to the development of type 2 diabetes. 11:27 By the time we see blood sugar rising, 11:29 we're now aware of a problem that has been 11:31 going on for quite some time. 11:34 In insulin resistance, what they figured out was, 11:38 it was in the 1990's when this was worked out 11:41 in great detail, they finally figured out what exactly is 11:44 going on in the cell that interferes with that process. 11:47 It interferes with the ability of insulin 11:50 to convey its signal to the cell. 11:52 And what they found out, of course, was actually a surprise. 11:55 It surprised me when I learned it. 11:58 What happens when insulin binds the receptor 12:01 on the outside, and then the receptor on the inside, 12:04 the same structure on the inside is supposed to send a signal, 12:07 that signal is interfered with somehow. 12:10 It's as though there's a roadblock 12:12 or some kind of impairment on the insulin receptor 12:15 on the inside of the cell. 12:16 And since the message isn't sent through the cell, 12:20 the cell doesn't ask glucose to come in. 12:23 Glucose isn't invited in, it doesn't come in. 12:26 Glucose, in that case, just builds up outside 12:29 the cell in the bloodstream. 12:31 This becomes what we all think of as type 2 diabetes. 12:35 But this underlying process of insulin resistance 12:38 has been going on for probably years, maybe decades, 12:42 prior to the development of type 2 diabetes. 12:45 Personally, I find roadblocks so frustrating. 12:49 I'm trying to get somewhere and a roadblock is holding me back. 12:53 The roadblock Dr. Racine referred to is 12:55 occurring inside of the cells. 12:57 The on-ramp for glucose to get into the cells is shut down. 13:01 And since glucose can't get off the freeway, 13:03 which is your blood vessels, because it can't get off the 13:06 freeway, congestion occurs. 13:08 We call it, high blood sugars stuck 13:11 because of the cellular roadblock. 13:13 What a mess. Can you picture it? 13:16 What's causing the roadblock? 13:18 Insulin resistance. 13:20 And just like freeway roadblocks, problems are 13:22 created in other locations. 13:24 And when it comes to our body, problems start brewing 13:27 in other places like the pancreas 13:29 where insulin is produced. 13:32 To control the blood sugars, the pancreas is having to kick out 13:36 more and more insulin. 13:38 And the cells literally become resistant to the insulin 13:43 so that you have to have more and more insulin. 13:44 And the pancreas gets to a place where it 13:46 can't put out any more. 13:48 We have receptors, insulin receptors that essentially 13:51 open up the gates for the cells to be flooded with glucose. 13:56 And you know, it's really picky. 13:57 We have a certain number of receptors on each cell, 14:01 and depending on their function, the amount of receptors differ. 14:05 When we are exposed to a lot of glucose molecules 14:09 in our circulation, the body essentially starts secreting 14:15 insulin to allow for each and every cell to consume glucose 14:19 as a source of energy. 14:20 However, when there's too much of it, it completely shuts down. 14:24 Or what happens is our body is not able to produce 14:28 enough insulin for that glucose to be consumed. 14:31 So not to go into a whole lot of chemistry, 14:34 but what happens is the body develops insulin resistance, 14:38 and the amount of insulin that is already in the system 14:42 cannot allow for this glucose to be used and to enter each cell. 14:46 So this means a lot of glucose molecules in the circulation. 14:50 And what happens is, when our tissue, whether it's the 14:53 capillaries or any cells that come in contact with that 14:57 incredible amount of glucose, they start getting damaged. 15:01 And so you see cascade of inflammation, oxidation, 15:05 oxidative stress causing the formation of 15:08 reaction oxygen species that go and damage the 15:11 walls of cells and tissue. 15:14 And that results in a lot of damage. 15:16 So there's an episode in, I Love Lucy, 15:19 which is an ancient TV program, where Lucy and Ethel have a 15:23 new job wrapping chocolates that are coming down a conveyor belt. 15:26 They're wearing these cute aprons and happily wrapping 15:29 each chocolate, until more and more and more chocolates 15:33 come down the conveyor belt, and they can't keep up. 15:36 Aagh! 15:38 They start stuffing the chocolates in their apron 15:39 pockets, in their mouths, and everything starts falling apart. 15:44 The result of insulin resistance in the body 15:47 and the eventual build-up of blood sugar 15:48 is not unwrapped chocolates. 15:50 It's ROS. 15:52 Remember him from a previous program? 15:54 Oxidation, inflammation, and damage; 15:56 that's what ROS leaves behind. 15:59 Let's take a look at the level of damage 16:01 that we're talking about. 16:03 Because if those blood sugars are off, 16:05 all kinds of things can occur. 16:09 And it's all kinds of diseases are affected 16:12 by blood sugar problems. 16:13 Type 2 diabetes where you have insulin resistance. 16:17 It could be circulation, it could be lung, 16:18 it could be cardiovascular, you name it. 16:22 Almost everything is involved with that. 16:25 Yeah, so insulin resistance clearly causes type 2 diabetes. 16:30 Meanwhile, it's causing a lot of other problems. 16:32 So most of us have heard of a disease that affects women 16:37 called, polycystic ovary syndrome. 16:40 Next to pregnancy, it's the most common cause of absent periods. 16:44 And so, it's a pathological state of absent periods. 16:46 Classically, it's a woman with months 16:49 and months between periods. 16:51 Usually heavy, but not always. 16:54 And with a higher risk of diabetes. 16:56 It turns out that insulin resistance is an important part 16:59 of the cause of polycystic ovary syndrome 17:01 happening in the ovaries. 17:04 Insulin resistance also increases one's risk for 17:06 heart disease and cancer. 17:09 It's closely related to obesity. 17:11 Obesity and insulin resistance probably have a kind of a 17:14 circular relationship where they promote each other, 17:17 and they both get worse the more obese we are. 17:21 Several cancers are known to be related to insulin resistance... 17:32 Cardiovascular diseases are related to insulin resistance. 17:35 Neurodegenerative diseases are also associated 17:39 with insulin resistance: 17:40 Alzheimer's and types of dementia that are involved with 17:45 insulin resistance in the brain. 17:48 Even ear, nose, and throat; a person who has type 2 diabetes 17:51 where their blood sugar is out of control, 17:53 you can't get infections under control. 17:55 What happens is, the high blood sugar is devastating 18:00 to really everything in the body. 18:01 Every cell in the body is damaged by high blood sugar. 18:04 Called glucose toxicity. 18:07 Even the pancreas has struggles and becomes 18:11 damaged by high blood sugars, 18:14 that causes more free radical damage. 18:17 So then, high blood sugars are really bad for the brain; 18:21 endomucin and everything. 18:23 Okay, then the high insulin levels is even worse 18:27 for their brain, and their heart. 18:28 You know, one of the number one causes 18:30 of heart attacks and strokes? 18:32 Insulin resistance as evidenced by excess insulin 18:35 floating through the bloodstream. 18:37 We actually tend to see the development of 18:40 inflammatory changes, plagues, hardening of the arteries, 18:44 which we see a lot on MRI images or the scans, 18:47 you know, of the carotid arteries, 18:50 resulting in vascular dementia, stroke, etcetera. 18:54 Whoa, wait a second. 18:56 Dr. Sherzai and Dr. Youngberg connected insulin resistance 19:00 with the number one killer in America. 19:02 Cardiovascular disease, heart attacks, strokes. 19:05 This is way bigger than just type 2 diabetes. 19:08 We were just provided a new piece of the puzzle. 19:11 A very crucial piece. 19:13 And that's not just high blood sugar that's the problem. 19:17 High insulin levels, not just high blood sugars, 19:20 are a major perpetrator of disease. 19:23 Putting on your detective hat, you can ask the doctor 19:25 to test your fasting blood insulin levels 19:28 before even blood sugars are off. 19:29 Lab results may reveal normal blood sugar levels 19:32 for years, even decades. 19:34 Dr. Racine mentioned that earlier. 19:36 Yet, insulin levels may be elevated, 19:39 signaling that there's a problem. 19:41 For instance, in insulin resistance or diabetes syndrome, 19:47 or even in cognitive concerns like Alzheimer's, 19:52 one of the main problems that drives cognitive decline 19:56 is insulin resistance that drives sugars high, 20:00 which then drives insulin high. 20:02 That's the main hormone we've got to fix, by the way. 20:05 And exercise is a key way to do that. 20:08 Once insulin levels go high, then what happens next? 20:11 After a while the blood sugars come down crashing too low, 20:15 and we get this reactive hypoglycemia 20:19 that then stresses out the adrenal gland, 20:22 because the amygdala of the brain is going like, 20:24 "Wow, blood sugars are crashing. 20:26 We're going to be shutting down." 20:28 The brain can't work without an appropriate amount of glucose. 20:31 And so, the amygdala basically forces the weak adrenals 20:36 that are already tired and don't want to do any work, 20:39 forces them to kick in really hardcore. 20:42 And now cortisol levels go from too low to too high 20:46 in a matter of just a few minutes. 20:49 And that swing in cortisol that goes up 20:53 really messes with our system. 20:55 Of course, it tells the liver to dump sugar. 20:57 That's the reason it's happening. 20:59 It's trying to bring the blood sugars back up. 21:01 But in doing so, we go through this rollercoaster 21:06 of hormonal fluctuation that involves too much insulin, 21:12 too little cortisol, too much sugar, too little sugar, 21:17 and then too much cortisol. 21:20 So we go through this rollercoaster 21:22 throughout the day, day after day. 21:24 That is really bad for the brain. 21:27 It's really bad for the immune system. 21:29 It's really bad for the cardiovascular system as well. 21:32 Now I used to love rollercoasters. 21:35 Emphasis on "used to." When I was young. 21:38 Now that I'm not so young, you couldn't pay me enough 21:41 to go on one. 21:42 We may not even be aware that we are on this 21:44 metabolic rollercoaster when we're young. 21:47 But in time, the body starts letting us know that it can't 21:51 tolerate the crashes and the force surges like it used to. 21:55 The body compensates for the highs and lows at first, 21:58 but over time it becomes a hot mess 22:01 that, as we've learned, is related to our weight, 22:04 obesity, to different cancers, heart attack, 22:07 stroke, hypoglycemia, 22:09 and neurodegenerative concerns like dementia. 22:13 In neurodegenerative conditions, insulin resistance plays a 22:17 major role because in certain circuits Alzheimer's disease 22:23 is actually tiled as type 3 diabetes because of the 22:26 predominance of insulin resistance. 22:30 And one thing that's important to understand 22:32 is that people always think that diabetes is bad, 22:34 but insulin resistance, which is a pre pre-diabetes stage 22:39 where the body has difficulty metabolizing glucose, 22:42 at that moment too people tend to have cognitive changes. 22:46 As a matter of fact, one of our researches showed that 22:49 in a large, very large nationwide sample of the 22:52 population, individuals who had insulin resistance 22:56 compared to those who didn't have insulin resistance 22:58 had lower cognitive state. 23:00 When they did neuropsychological testing on them, they actually 23:02 scored poorly. 23:04 So obviously this can advance to pre-diabetes and diabetes, 23:08 but insulin resistance is a very important concept. 23:12 And it's something that is reversible. 23:14 Neurodegenerative diseases are also associated 23:18 with insulin resistance. 23:20 Alzheimer's and types of dementia that are involved with 23:24 insulin resistance in the brain. 23:27 And there's some evidence that high blood pressure 23:29 is also related to insulin resistance. 23:31 So yes, it causes a lot of problems on the way 23:33 to developing type 2 diabetes. 23:35 And that high insulin now has to be broken down. 23:42 Otherwise, we would go into coma with 23:44 super low blood sugars, right? 23:45 But did you know that the very enzyme called, 23:49 Insulin Degrading Enzyme, IDE, so the same enzyme that 23:57 breaks down insulin, appropriately so, 24:00 is also the one that removes beta-amyloid plague 24:02 from the brain? 24:04 And so, if somebody is in a constant state 24:08 of hyperinsulinemia, excess production of insulin, 24:13 which happens years and decades before the full diagnosis 24:17 of diabetes, or even pre-diabetes in many people. 24:20 This is fascinating. 24:22 Elevated insulin levels may be keeping blood sugar levels 24:25 down, but having to deal with the elevated insulin level 24:29 costs the body a lot. 24:31 Elevated insulin levels lead to sex hormone imbalance 24:34 where estrogens and testosterone don't have a 24:37 healthy ratio with each other. 24:39 It affects immune function, promotes weight gain, 24:41 and cancers, inflammation. 24:43 And it worsens insulin resistance. 24:46 And here we just learned that it can distract the body 24:49 from clearing out toxins from the brain, 24:51 these beta-amyloid plagues that are associated with 24:54 dementia and Alzheimer's. 24:56 Wow. 24:58 Insulin resistance is a very important concept. 25:00 And it's something that is reversible. 25:03 So let's recap. 25:06 There's been so much information today. 25:08 What did we learn from our investigation into this topic? 25:11 First, we saw some shocking statistics that this problem 25:15 is a whole lot more relative than we might have thought. 25:19 Diabetes is not just a disease for a specific group of people. 25:24 It's affecting young and old, overweight, normal weight, 25:28 people who eat a lot of sugar, 25:30 and people who don't eat a lot of sugar. 25:33 We learned that insulin resistance is a major mechanism 25:36 that is driving the body's inability to process blood sugar 25:40 like it once did. 25:42 Insulin was defined as the polite gentleman, 25:45 or Oregonian gas attendant, that opens the cell up 25:49 for blood glucose to enter in. 25:51 And just to avoid confusion, blood glucose and blood sugar, 25:55 they're the same thing. 25:56 You can use the terms interchangeably. 25:58 Do you remember what we likened your blood glucose to? 26:01 It's the fuel. The gasoline of your body. 26:04 Once it enters the cell, it is used to make energy 26:07 for the cell to function optimally. 26:09 We all want good energy production. 26:12 Then we learned that the first signs of insulin resistance 26:15 aren't necessarily elevated blood sugar levels. 26:18 No, they can be normal for years. 26:21 You go to the doctor, get your yearly check-up, 26:23 your fasting blood sugars are normal. 26:25 Good to go. 26:26 But maybe you have elevated blood cholesterol 26:29 or triglycerides. 26:30 Or you're gaining weight or blood pressure is higher. 26:33 Or you've developed PCOS. 26:35 In other words, there are other clues that insulin resistance 26:39 is simmering in the background. 26:41 Insulin resistance leads to elevated insulin levels 26:44 in the blood; and this is what can drive 26:46 a lot of these imbalances. 26:49 The jaw-dropping part for me was how these elevated insulin 26:53 levels can also affect brain health 26:55 and increase our risk of neurodegenerative diseases. 27:00 Are you scared? 27:01 It would be hard not to be with the realization 27:03 that nearly 50% of us are experiencing this. 27:07 Except that there are a whole lot of other words 27:10 that begin with "R" other than "resistance." 27:13 Like, reversible, renewable, recoverable, 27:18 restoration, redemption. 27:21 These are powerful words that I believe 27:24 every cell of our body has the ability to experience. 27:28 They describe what is possible for you. 27:31 Evidence that you were made for health. |
Revised 2025-03-03